The most common cardiac arrhythmia, atrial fibrillation, occurs when the normal electrical impulses that are generated by the SA node are overwhelmed by disorganized electrical impulses in the atria.
These disorganized impulses cause the muscles of the upper chambers of the heart to quiver (fibrillate) and this leads to the conduction of irregular impulses to the ventricles.
For ACLS, atrial fibrillation becomes a problem when the fibrillation produces a rapid heart rate which reduces cardiac output and causes symptoms or an unstable condition.
When atrial fibrillation occurs with a (RVR) rapid ventricular rate (rate > 100 beats/min), this is called a tachyarrhythmia. This tachyarrhythmia may or may not produce symptoms. Significant symptoms that occur are due to a reduction in cardiac output.
The following is a list of the most common symptoms.
- palpitations or chest discomfort
- shortness of air and possibly respiratory distress
- hypotension, light-headedness and possibly loss of consciousness
- peripheral edema, jugular vein distention, and possibly pulmonary edema
For the purpose of ACLS, it is important to be able to recognize atrial fibrillation when the patient is symptomatic. On an ECG monitor, there are two major characteristics that will help you identify atrial fibrillation.
- No p-waves before the QRS on the ECG. This is because there are no coordinated atrial contractions.
- The heart rate will be irregular. Irregular impulses that the ventricles are receiving cause the irregular heart rate.
When the heart rate is extremely rapid, it may be difficult to determine if the rate is irregular, and the absence of p-waves will be the best indicator of atrial fibrillation.
ACLS Treatments:
For the purposes of ACLS atrial fibrillation is treated when the arrhythmia/tachyarrhythmia produces hemodynamic instability and serious signs and symptoms.
For the patient with unstable tachycardia due to a tachyarrhythmia, immediate cardioversion is recommended. Drugs are not used to manage unstable tachycardia. The appropriate voltage for cardioverting unstable atrial fibrillation is 120-200 J.
Cardioversion of stable atrial fibrillation should be performed with caution if the arrhythmia is more than 48 hours old and no anticoagulant therapy has been initiated due to the risk of emboli that can cause MI and stroke.
Below is a short video which will help you quickly identify atrial fibrillation on a monitor.
Please allow several seconds for the video to load. (5.11 mb)
Click for next Rhythm Review: Other Tachycardias
Top Questions Asked On This Page
Q: To treat a patient with rapid atrial fibrillation (HR >180bpm), can calcium channel blockers be given to help control the rate?
A: Yes, in some cases, calcium channel blockers are a good choice to help control atrial fibrillation with a rapid ventricular rate.
Q: What are atrial fibrillation and atrial flutter considered in terms of narrow or wide QRS complexes?
A: Unless there is an underlying block the QRS complex will typically be normal/narrow. There is the possibility of occasional wide QRS complexes due to abnormal ventricular depolarization via an accessory pathway.
Q: What does the DC before the word cardioversion stand for?
A: DC cardioversion simply means direct current cardioversion.
Click to view more top questions
EK says
August 26, 2018 at 10:09 amHi There
If a patient with Atrial fibrillation suddenly lose pulse, do you follow ACLS protocol which mean this is a PEA, start CPR, Epi… and you do not
Cardiovert
OR do you still consider this unstable and try to cardiovert first?
Jeff with admin. says
August 26, 2018 at 11:50 amIf they lose consciousness and there is no palpable pulse then you would treat this as PEA.
If you can not palpate a pulse but the patient remains conscious then you can be assured that they do have a pulse.
In this case, you would want to determine if the patient is unstable. If the patient is unstable with atrial fibrillation then synchronized cardioversion would be the treatment of choice. There are risks involved with providing synchronized cardioversion to a patient with atrial fibrillation if they are not anticoagulated. There is a risk of stroke and it would be wise to rule out thrombus prior to cardioversion if possible. Thrombus can be ruled out with a transesophageal echocardiogram.
Kind regards,
Jeff
Rose says
December 1, 2017 at 5:07 pmCan I apply this for nclex RN exam?
Jeff with admin. says
December 3, 2017 at 9:41 amI don’t think that either of the courses offered on this website would be very beneficial for preparing for NCLEX.
The ACLS and PALS courses offered on this website are designed to help people prepare for the American Heart Association ACLS and PALS provider course.
Kind regards,
Jeff
Ashley Heard says
September 12, 2017 at 9:47 pmHI Jeff,
I am finding this website to be a great resource. Thank you!
Interpreting ECGs is not part of my job so that is the portion that I am experiencing the most difficulty with. Memorizing and understanding the medications and algorithms is much easier for me.
Can you provide some guidance on how to recognize the difference between atrial and ventricular on an ECG?
Also, how can you know but the ECG alone is the rhythm is pulseless?
Thanks so much for your help!!
Jeff with admin. says
September 14, 2017 at 5:45 amThe ventricular portion of the ECG is known as the QRS complex. The QRS complex is the electrical equivalent of the ventricular contraction. If there is no ventricular contraction, there is no pulse. Therefore, if your patient has a pulse, you will see a QRS complex. The QRS complex will be the tallest and most pronounced portion of the ECG reading. The atrial impulses may or may not be present depending on the rhythm. Atrial impulses are recognized on the ECG monitor as P-waves that typically precede the QRS complex. The p-waves are much smaller than the QRS complexes.
A rhythm can be determined to be pulseless simply by manually palpating for a pulse. You cannot determine a pulseless state by only looking at the ECG monitor.
Kind regards,
Jeff
Phil says
August 15, 2017 at 7:35 pmJeff,
What are the rhythms that cardioversion is the first thing that must be done? I mean what shall a beginner like me should see or look that once I see it, it will tell me to perform cardioversion right away? Does it make sense?
Thanks,
Phil
Jeff with admin. says
August 17, 2017 at 2:20 amIf you see a patient that is hemodynamically unstable with a wide complex tachycardia, this would be an indication for immediate cardioversion. In fact, any patient that is unstable with a tachyarrhythmia should be prepared for cardioversion. There is a difference between being unstable and symptomatic and knowing that difference is important. Three of the most important signs of instability are changes in the level of consciousness, SBP < 90, and unrelieved chest pain.Kind regards, Jeff
Rachel Jolokai says
November 9, 2017 at 3:51 pmCan you further elaborate on difference between symptomatic vs unstable?
Jeff with admin. says
November 13, 2017 at 11:26 amThe major difference that will be associated with unstable arrhythmias will be a systolic blood pressure less than 90 mmHg with other signs of poor perfusion.
A patient that is simply symptomatic may have dizziness, tachycardia, weak pulses, etc. but the systole blood pressure remains greater than 90 mmHg, and there are no LOC changes or other signs of poor perfusion.
I hope that makes sense.
Kind regards,
Jeff
LouiseAW says
February 16, 2017 at 6:43 pmwhy the voltage difference in the Cardioversion of Fib Flutter ?
Jeff with admin. says
February 20, 2017 at 5:18 amThe reason is that atrial flutter tends to convert with a much lower dose of electricity than atrial fibrillation. Kind regards, Jeff
orangele says
January 19, 2017 at 11:04 pmSo it is mentioned that blot clots must be ruled out prior to cardioversion for Atrial Fibrillation, but that cardioversion is indicated if the patient is unstable. So therefore I assume that echo is preferred but once patient is unstable that cardioversion must be performed without echo?
Jeff with admin. says
January 20, 2017 at 7:28 pmThat is correct if a patient has reached the point where there is no other way to keep them stable and cardioversion is indicated then cardioversion may be performed emergently.
I think that this would be a rare occurrence. Usually instability with atrial fibrillation is caused by a rapid heart rate. Typically, you can apply some type of rate control medicaton like cardizem or metoprolol to bring the heart rate down. There are a lot of variables and a lot of options and cardioversion for unstable atrial fibrillation without an TEE would be the last option.
Regards,
Jeff
Nursey54 says
January 14, 2017 at 11:39 pmI have to give your course 5 stars! I use it every time I recert for my PACU job and do extremely well.
I plan to do PALS next month. Do you have a course for PALS?
Thanks ! Alice
Monica says
December 16, 2016 at 2:29 pmWhat about this scenario:
82 y/o m pale, cool diaphoretic. No c/o pain and negative on Stroke scale. Pt vs 84/43. 71HR 32R 90%. BGL 63. Pmhx DM HTN MI AFIB CHF HYPERLIPIDEMIA. oral glucose is given and bgl is 224. 12 lead shows AFIB @71 RBBB SUBSEQUENT 12 leads are same with no changes. Rate is between 69 and 102. Cardizem is contra-indicated due to hypotension. No flight is available and closest ER is 1 hour.
Jeff with admin. says
December 18, 2016 at 10:47 amGive an anticoagulant to prevent thrombus. Give Epinephrine or dopamine IV infusion to control hypotension. Transport the patient by EMS to the nearest ICU for evaluation.
Kind regards,
Jeff
Patrick says
January 3, 2017 at 4:27 pmWould norepi not be a better choice to avoid the positive chronotropic effect?
Jeff with admin. says
January 4, 2017 at 4:07 amIf there were a problem with rate control, you could use norepinephrine. Since the rate is in the 70’s. Dopamine or epinephrine would be acceptable.
There are two other possibilities with this scenario. NSTEMI or sepsis. It would be wise to check a white count. If dopamine is started and the patient develops any chest pain, stop the dopamine and suspect NSTEMI. Norepinephrine would be the best choice if this happens.
Kind regards,
Jeff
abosuyonov says
September 28, 2016 at 5:27 amThank you .
Just to clarify , even though the ACLS algorithm states that for Unstable Afib we perform synchronized cardioversion, however in reality it appears that for unstable a fib, we stabilize with medications, and once a patient is stable, we cardiovert after doing a TEE. is that correct?
Why don’t we in real life scenario not perform synchronize cardioversion for unstable Afib as it states in ACLS algorithm?
Jeff with admin. says
September 28, 2016 at 11:06 amThere is a gray area between stable and unstable with regard to tachycardias like afib and aflutter. If there is no other way of reversing or correcting the unstable condition and cardiac arrest is eminent then synchronized cardioversion is necessary and should be done even if TEE has not been performed.
Kind regards,
Jeff
abosuyonov says
September 27, 2016 at 1:26 pmHello Jeff.
Just to clarify .
For unstable Afib we perform synchronized cardioversion . How can one perform TEE prior to that if pt is symptomatic . It’ll really be almost impossible to do TEE with the symptoms he us experiencing ?
Jeff with admin. says
September 28, 2016 at 4:11 amI personally have never seen a case of unstable afib that needed immediate cardioversion. I have seen some cases of unstable afib that responded well to rate controlling medications, blood pressure infusions, and anticoagulation. After the patient was stabilized with IV medications the TEE is performed and then cardioversion if necessary.
Lets say that a patient is unstable with afib and then has an MI and goes into cardiac arrest (VF). You would take your chances and perform defibrillation. The risks involved in to defibrillating are greater than the risks of thrombus from afib. Hope that makes sense.
Kind regards,
Jeff
Allison says
September 22, 2016 at 8:18 pmGotta say I am taking my recertification tomorrow . I wish I found this sight earlier. Have been looking at it all day. You have really done a great job . Thanks ?